Research on Multiple Chemical Sensitivity ( MCS )

نویسندگان

  • Anne C. Steinemann
  • Amy L. Davis
چکیده

This document lists citations for peer-reviewed journal articles that support a physiological basis for MCS. Disruption of the blood-brain barrier and neuronal cell death in cingulate cortex, dentate gyrus, thalamus, and hypothalamus in a rat model of Gulf-War syndrome. combined exposure to low doses of pyridostigmine bromide, DEET, and permethrin produce neurochemical and neuropathological alteration in cerebral cortex, hippocampus, and cerebellum. 1996. Increased neurotoxicity following concurrent exposure to pyridostigmine bromide, DEET, and chlorpyrifos. Uranyl acetate-induced sensorimotor deficit and increased nitric oxide generation in the central nervous system in rats. W.A. 2004. Co-exposure to pyridostigmine bromide, DEET, and/or permethrin causes sensorimotor deficit and alterations in brain acetylcholinesterase activity. Pharmacology, Biochemistry, and Behavior 77(2): 253-62. Abu-Qare A.W., Abou-Donia M.B. 2001. Combined exposure to sarin pyridostigmine bromide increased levels of rat urinary 3-nitrotyrosine and 8-hydroxy-2'deoxyguanosine, biomarkers of oxidative stress. Toxicology Letters 123(1): 51-58.

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تاریخ انتشار 2009